Search results for "Cellular microbiology"

showing 10 items of 11 documents

Transcytosis of Bacillus subtilis extracellular vesicles through an in vitro intestinal epithelial cell model

2020

Bacterial EVs have been related to inter-kingdom communication between probiotic/pathogenic bacteria and their hosts. Our aim was to investigate the transcytosis process of B. subtilis EVs using an in vitro intestinal epithelial cell model. In this study, using Confocal Laser Scanning Microscopy, we report that uptake and internalization of CFSE-labeled B. subtilis EVs (115 nm ± 27 nm) by Caco-2 cells are time-dependent. To study the transcytosis process we used a transwell system and EVs were quantified in the lower chamber by Fluorescence and Nanoparticle Tracking Analysis measurements. Intact EVs are transported across a polarized cell monolayer at 60–120 min and increased after 240 min …

0301 basic medicineCell Survivalmedia_common.quotation_subjectNanoparticle tracking analysislcsh:MedicineBacillus subtilisCellular imagingmedicine.disease_causeModels BiologicalGastrointestinal epitheliumArticleEpithelium//purl.org/becyt/ford/1 [https]Extracellular Vesicles03 medical and health sciences0302 clinical medicineFunctional FoodmedicineHumansCellular microbiology//purl.org/becyt/ford/1.6 [https]Internalizationlcsh:ScienceCell Proliferationmedia_commonMicroscopy ConfocalMultidisciplinarybiologyChemistryProbioticslcsh:RCell PolarityEpithelial CellsPathogenic bacteriaExtracellular vesiclesbiology.organism_classificationGITIn vitroEpitheliumCell biologyIntestines030104 developmental biologymedicine.anatomical_structureTranscytosis030220 oncology & carcinogenesislcsh:QCaco-2 CellsTranscytosisBacillus subtilisScientific Reports
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Encapsulation mechanisms and structural studies of GRM2 bacterial microcompartment particles

2019

Bacterial microcompartments (BMCs) are prokaryotic organelles consisting of a protein shell and an encapsulated enzymatic core. BMCs are involved in several biochemical processes, such as choline, glycerol and ethanolamine degradation and carbon fixation. Since non-native enzymes can also be encapsulated in BMCs, an improved understanding of BMC shell assembly and encapsulation processes could be useful for synthetic biology applications. Here we report the isolation and recombinant expression of BMC structural genes from the Klebsiella pneumoniae GRM2 locus, the investigation of mechanisms behind encapsulation of the core enzymes, and the characterization of shell particles by cryo-EM. We …

0301 basic medicineKlebsiella pneumoniaeScience030106 microbiologyGeneral Physics and AstronomyLyasesGeneral Biochemistry Genetics and Molecular BiologyArticleCholine03 medical and health sciencesSynthetic biologyBacterial ProteinsBacterial microcompartmentCryoelectron microscopyOrganellelcsh:ScienceCellular microbiologychemistry.chemical_classificationOrganellesBacterial structural biologyMultidisciplinarybiologyChemistryStructural geneQSignal transducing adaptor proteinGeneral ChemistryLyasebiology.organism_classificationRecombinant ProteinsKlebsiella pneumoniae030104 developmental biologyEnzymeGenetic LociBiophysicslcsh:QSynthetic BiologyNature Communications
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Chromatin organization regulates viral egress dynamics.

2017

Various types of DNA viruses are known to elicit the formation of a large nuclear viral replication compartment and marginalization of the cell chromatin. We used three-dimensional soft x-ray tomography, confocal and electron microscopy, combined with numerical modelling of capsid diffusion to analyse the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the transport of progeny viral capsids to the nuclear envelope. Our data showed that the formation of the viral replication compartment at late infection resulted in the enrichment of heterochromatin in the nuclear periphery accompanied by the compaction of chromatin. Random walk modelling of herpes s…

0301 basic medicineX-RAY TOMOGRAPHYvirusesmedicine.disease_cause2.2 Factors relating to physical environmentHistoneschemistry.chemical_compoundMiceINFECTION2.2 Factors relating to the physical environmentREPLICATION COMPARTMENTSAetiologyVirus ReleaseMicroscopyMultidisciplinaryMicroscopy ConfocalQRMICROSCOPYChromatin3. Good healthChromatinCell biologyTIMEOther Physical Sciencesmedicine.anatomical_structureInfectious DiseasesCapsidConfocalMedicineFemaleInfectionVESICLE FORMATIONNUCLEAR ARCHITECTUREHeterochromatinScienceBiology114 Physical sciencesArticleCell Line03 medical and health sciencesmedicineHerpes virusAnimalsCellular microbiologyNuclear export signalcell chromatinCell NucleusHERPES-SIMPLEX-VIRUSBiological TransportVirology030104 developmental biologyHerpes simplex viruschemistryViral replicationCELLS1182 Biochemistry cell and molecular biologyBiochemistry and Cell BiologyDNA virusesNucleusDNABiomarkersHISTONE MODIFICATIONSVirus Physiological PhenomenaScientific reports
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Type III Secretion-Dependent Cell Cycle Block Caused in HeLa Cells by Enteropathogenic Escherichia coliO103

2001

ABSTRACT Rabbit enteropathogenic Escherichia coli (EPEC) O103 induces in HeLa cells an irreversible cytopathic effect characterized by the recruitment of focal adhesions, formation of stress fibers, and inhibition of cell proliferation. We have characterized the modalities of the proliferation arrest and investigated its underlying mechanisms. We found that HeLa cells that were exposed to the rabbit EPEC O103 strain E22 progressively accumulated at 4C DNA content and did not enter mitosis. A significant proportion of the cells were able to reinitiate DNA synthesis without division, leading to 8C DNA content. This cell cycle inhibition by E22 was abrogated in mutants lacking EspA, -B, and -D…

G2 Phase[SDV]Life Sciences [q-bio]ImmunologyCyclin BMitosisReceptors Cell SurfacePATHOGENICITECyclin BMicrobiology03 medical and health sciencesBacterial ProteinsCDC2 Protein KinaseEscherichia coliHumansCyclin B1PhosphorylationCyclin B1Adhesins BacterialMitosisCytoskeleton030304 developmental biologyIntimin0303 health sciencesCyclin-dependent kinase 1Cellular Microbiology: Pathogen-Host Cell Molecular Interactionsbiology030306 microbiologyCell growthEscherichia coli ProteinsCell CycleREARRANGEMENTCell cycle[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyCell biology[SDV] Life Sciences [q-bio]Infectious Diseasesbiology.proteinTyrosineParasitologyCarrier ProteinsCDC2 Protein KinaseBacterial Outer Membrane ProteinsHeLa Cells
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Novel Paraoxonase 2-Dependent Mechanism Mediating the Biological Effects of the Pseudomonas aeruginosa Quorum-Sensing Molecule N-(3-Oxo-Dodecanoyl)-l…

2015

ABSTRACT Pseudomonas aeruginosa produces N -(3-oxo-dodecanoyl)- l -homoserine lactone (3OC12), a crucial signaling molecule that elicits diverse biological responses in host cells thought to subvert immune defenses. The mechanism mediating many of these responses remains unknown. The intracellular lactonase paraoxonase 2 (PON2) hydrolyzes and inactivates 3OC12 and is therefore considered a component of host cells that attenuates 3OC12-mediated responses. Here, we demonstrate in cell lines and in primary human bronchial epithelial cells that 3OC12 is rapidly hydrolyzed intracellularly by PON2 to 3OC12 acid, which becomes trapped and accumulates within the cells. Subcellularly, 3OC12 acid acc…

ImmunologyBlotting WesternHomoserineMitochondrionMicrobiologyCell LineHost-Parasite Interactionschemistry.chemical_compoundLactonesLactonaseHomoserineHumansImmunoprecipitationPseudomonas InfectionsChromatography High Pressure LiquidCellular Microbiology: Pathogen-Host Cell Molecular InteractionsMicroscopy ConfocalbiologyKinaseAryldialkylphosphataseQuorum SensingQuorum sensingCytosolInfectious DiseasesBiochemistrychemistryPseudomonas aeruginosabiology.proteinPhosphorylationParasitologyRNA InterferenceIntracellular
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Virulent synergistic effect between Enterococcus faecalis and Escherichia coli assayed by using the Caenorhabditis elegans model.

2008

5 pages; International audience; BACKGROUND: The role of enterococci in the pathogenesis of polymicrobial infections is still debated. The purpose of this study was to evaluate the effect of virulent enterococci in the presence or absence of Escherichia coli strains in the in vivo Caenorhabditis elegans model. PRINCIPAL FINDINGS: This study demonstrated that there was a synergistic effect on virulence when an association of enterococci and E. coli (LT50 = 1.6 days+/-0.1 according to the tested strains and death of nematodes in 4 days+/-0.5) was tested in comparison with enterococci alone (LT50 = 4.6 days+/-0.1 and death in 10.4 days+/-0.6) or E. coli alone (LT50 = 2.1+/-0.9 and deaths 6.6+/…

MESH : Virulence FactorsInfectious Diseases/Gastrointestinal InfectionsMESH : Escherichia colilcsh:MedicineMESH : Genotypemedicine.disease_causeMESH: Regression AnalysisPathogenesisMESH: GenotypeInfectious Diseases/Bacterial InfectionsMESH : Regression AnalysisGenotype[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisEnterococcus faecalis[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: AnimalsMESH : Anti-Bacterial AgentsMESH : Enterococcus faecalislcsh:ScienceCaenorhabditis elegans0303 health sciencesMultidisciplinarybiologyMESH: Escherichia coliBacterial Infections3. Good healthAnti-Bacterial AgentsMicrobiology/Immunity to InfectionsMESH : Bacterial InfectionsGastroenterology and Hepatology/Gastrointestinal Infections[SDV.IMM]Life Sciences [q-bio]/ImmunologyRegression AnalysisMicrobiology/Cellular Microbiology and PathogenesisResearch ArticleMESH: Enterococcus faecalis[SDV.IMM] Life Sciences [q-bio]/ImmunologyGenotypeMESH: Bacterial InfectionsVirulence FactorsVirulenceEnterococcus faecalisMicrobiologyMESH : Caenorhabditis elegans03 medical and health sciencesIn vivoMESH: Anti-Bacterial AgentsMESH: Caenorhabditis elegansmedicineEscherichia coliAnimalsCaenorhabditis elegansEscherichia coli030304 developmental biologyMESH: Virulence Factors030306 microbiologylcsh:RMicrobiology/Medical Microbiology[SDV.EE.IEO] Life Sciences [q-bio]/Ecology environment/Symbiosisbiology.organism_classificationMESH : Disease Models AnimalDisease Models AnimalEnterococcuslcsh:QMESH : AnimalsMESH: Disease Models Animal[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisPloS one
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Truncated internalin A and asymptomatic Listeria monocytogenes carriage: in vivo investigation by allelic exchange

2004

ABSTRACT Allelic exchange of the region coding for the C terminus of InlA between one epidemic (with an 80-kDa InlA) and one asymptomatic (with a 47-kDa InlA) carriage Listeria monocytogenes strain confirmed the need for this region for internalin entry in vitro. Interestingly, restoration of internalin A functionality did not result in full virulence in chicken embryo assays.

Molecular Sequence DataImmunologyVirulenceChick Embryomedicine.disease_causeMicrobiologyMicrobiology03 medical and health sciencesBacterial ProteinsListeria monocytogenesIn vivomedicineAnimalsHumansInternalinAlleleAlleles030304 developmental biology0303 health sciencesCellular Microbiology: Pathogen-Host Cell Molecular InteractionsBase SequenceVirulencebiology030306 microbiologyMicrobiology and Parasitologybacterial infections and mycosesbiology.organism_classificationListeria monocytogenesVirologyMicrobiologie et ParasitologieIn vitro3. Good healthInfectious DiseasesCarriage[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyParasitologyCaco-2 CellsBacteria
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AP-1 Transcription Factor Serves as a Molecular Switch between Chlamydia pneumoniae Replication and Persistence

2015

ABSTRACT Chlamydia pneumoniae is a Gram-negative bacterium that causes acute or chronic respiratory infections. As obligate intracellular pathogens, chlamydiae efficiently manipulate host cell processes to ensure their intracellular development. Here we focused on the interaction of chlamydiae with the host cell transcription factor activator protein 1 (AP-1) and its consequence on chlamydial development. During Chlamydia pneumoniae infection, the expression and activity of AP-1 family proteins c-Jun, c-Fos, and ATF-2 were regulated in a time- and dose-dependent manner. We observed that the c-Jun protein and its phosphorylation level significantly increased during C. pneumoniae development.…

Small interfering RNAGene knockdownCellular Microbiology: Pathogen-Host Cell Molecular InteractionsTranscription GeneticImmunologyChlamydiaeGene Expression Regulation BacterialHep G2 CellsChlamydophila pneumoniaeBiologybiology.organism_classificationMicrobiologyBacterial LoadMicrobiologyTranscription Factor AP-1AP-1 transcription factorInfectious DiseasesTranscription (biology)Host-Pathogen InteractionsHepatocytesHumansPhosphorylationParasitologyTranscription factorIntracellularInfection and Immunity
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Subcytocidal attack by staphylococcal alpha-toxin activates NF-kappaB and induces interleukin-8 production.

2001

ABSTRACTFormation of transmembrane pores by staphylococcal alpha-toxin can provoke a spectrum of events depending on target cell species and toxin dose, and in certain cases, repair of the lesions has been observed. Here, we report that transcriptional processes are activated as a response of cells to low toxin doses. Exposure of monocytic (THP-1) or epithelial (ECV304) cells to 40 to 160 ng/ml alpha-toxin provoked a drop in cellular ATP level that was followed by secretion of substantial amounts of interleukin-8 (IL-8). Cells transfected with constructs comprising the proximal IL-8 promoter fused to luciferase or to green fluorescent protein cDNA exhibited enhanced reporter gene expression…

StaphylococcusImmunologyBacterial ToxinsBiologymedicine.disease_causeMicrobiologyCell LineHemolysin ProteinsAdenosine TriphosphatemedicineHumansSecretionLuciferaseInterleukin 8Promoter Regions GeneticRegulation of gene expressionReporter geneCellular Microbiology: Pathogen-Host Cell Molecular InteractionsToxinInterleukin-8NF-kappa BTransfectionMolecular biologyInfectious DiseasesCell cultureParasitologyCaltech Library ServicesInfection and immunity
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Chlamydia trachomatis Infection and Anti-Hsp60 Immunity: The Two Sides of the Coin

2009

Chlamydia trachomatis (CT) infection is one of the most common causes of reproductive tract diseases and infertility. CT-Hsp60 is synthesized during infection and is released in the bloodstream. As a consequence, immune cells will produce anti-CT-Hsp60 antibodies. Hsp60, a ubiquitous and evolutionarily conserved chaperonin, is normally sequestered inside the cell, particularly into mitochondria. However, upon cell stress, as well as during carcinogenesis, the chaperonin becomes exposed on the cell surface (sf-Hsp60) and/or is secreted from cells into the extracellular space and circulation. Reports in the literature on circulating Hsp and anti-Hsp antibodies are in many cases short on detai…

lcsh:Immunologic diseases. Allergyanimal structuresImmunologyCardiovascular Disorders/Heart FailurePublic Health and Epidemiology/Infectious DiseasesChlamydia trachomatisPathology/Immunologychemical and pharmacologic phenomenaReviewmedicine.disease_causecomplex mixturesMicrobiologyAutoimmune DiseasesInfectious Diseases/Bacterial InfectionsPathogenesisImmune systemImmunityVirologyGeneticsmedicineAnimalsHumansImmunology/Cellular Microbiology and Pathogenesislcsh:QH301-705.5Molecular BiologyRheumatology/Autoimmunity Autoimmune and Inflammatory DiseasesAntigens BacterialbiologySettore BIO/16 - Anatomia UmanaMultiple sclerosisfungiAutoantibodyChaperonin 60Chlamydia Infectionsmedicine.diseaseHSP60 ChlamydiaMicrobiology/Immunity to Infectionslcsh:Biology (General)Immunologybiology.proteinParasitologyHSP60AntibodyDiabetes and Endocrinology/Type 1 Diabeteslcsh:RC581-607Chlamydia trachomatisPLoS Pathogens
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